Tag Archives: Intelligent Design

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How biological convergence falsifies Darwinian evolution

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Cornelius Hunter, a software engineer / biologist with a Ph.D in bioinformatics from UIUC explains the latest discovery of biological convergence on his blog. (H/T Tweet from J. Warner Wallace)

Excerpt:

The theory of evolution states that the species arose spontaneously, one from another via a pattern of common descent. This means the species should form an evolutionary tree, where species that share a recent common ancestor, such as two frog species, are highly similar, and species that share a distant common ancestor, such as humans and squids, are very different. But the species do not form such an evolutionary tree pattern. In fact this expectation has been violated so many times it is difficult to keep track. These violations are not rare or occasional anomalies, they are the rule. Entire volumes have been written on them. Many examples are the repeated designs found in what, according to evolution, must be very distant species. Such evolutionary convergence is biology’s version of lightning striking twice. To explain this evolutionists must say that random mutations just happened to hit upon the same detailed, intricate design at different times, in different parts of the world, in different ecological niches, and so forth. The idea that the most complex designs we know of would spontaneously arise by themselves is, itself, not scientifically motivated and a real stretch of the imagination. But for the same intricate designs to arise independently by chance is even more of a stretch. That is why evolutionist’s claim this week that they have found evidence for convergent evolution was so intriguing.

[…]Though evolutionists sometimes deny biological convergence, it is a scientific fact. And a paper from this week added yet another example:

In mammals, hearing is dependent on three canonical processing stages: (i) an eardrum collecting sound, (ii) a middle ear impedance converter, and (iii) a cochlear frequency analyzer. Here, we show that some insects, such as rainforest katydids, possess equivalent biophysical mechanisms for auditory processing. Although katydid ears are among the smallest in all organisms, these ears perform the crucial stage of air-to-liquid impedance conversion and signal amplification, with the use of a distinct tympanal lever system. Further along the chain of hearing, spectral sound analysis is achieved through dispersive wave propagation across a fluid substrate, as in the mammalian cochlea. Thus, two phylogenetically remote organisms, katydids and mammals, have evolved a series of convergent solutions to common biophysical problems, despite their reliance on very different morphological substrates.

It is another curious example of biological convergence, so rather than attempt to deny the undeniable, evolutionists now claim it as another confirmation of evolution.

I’m a software engineer, and we re-use components all the time for different programs that have no “common ancestor”. E.g. – I can develop my String function library and use it in my web application and my Eclipse IDE plug-in, and those two Java programs have no common ancestry, but they do have a common designer. So you find the same bits in two different programs because I am the developer of both programs.

Previously, I blogged about another example of convergence reported by Science Daily. One of the predictions of intelligent design theory is that examples of convergence, which is really just re-use of common code by the designer, will be everywhere in nature. And that predictions just keeps getting confirmed as science marches forward, and the primitive religion of naturalism retreats.

News

New paper finds that mass of asteroid belts affect habitability of planets

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Science Daily reports. (H/T Evolution News via ECM)

Excerpt:

They suggest that the size and location of an asteroid belt, shaped by the evolution of the Sun’s protoplanetary disk and by the gravitational influence of a nearby giant Jupiter-like planet, may determine whether complex life will evolve on an Earth-like planet.

This might sound surprising because asteroids are considered a nuisance due to their potential to impact Earth and trigger mass extinctions. But an emerging view proposes that asteroid collisions with planets may provide a boost to the birth and evolution of complex life.

Asteroids may have delivered water and organic compounds to the early Earth. According to the theory of punctuated equilibrium, occasional asteroid impacts might accelerate the rate of biological evolution by disrupting a planet’s environment to the point where species must try new adaptation strategies.

The astronomers based their conclusion on an analysis of theoretical models and archival observations of extrasolar Jupiter-sized planets and debris disks around young stars. “Our study shows that only a tiny fraction of planetary systems observed to date seem to have giant planets in the right location to produce an asteroid belt of the appropriate size, offering the potential for life on a nearby rocky planet,” said Martin, the study’s lead author. “Our study suggests that our solar system may be rather special.”

There’s a long list of factors that have to be present for a planet to support life – and more keep appearing every day. Here’s another recent one from the journal Nature.

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Paul Nelson: the most interesting and significant paper we’ve read in years

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Wow, check out this post by Paul Nelson over at Evolution News.

Excerpt:

Now, the paper I retrieved for my co-worker, entitled “The Levinthal paradox of the interactome,” Protein Science 20 (2011):2074-79, explains why the space of “being alive” is so much vastly smaller, and harder to find, than the space of being “not alive.” The paper is short (only six pages) and was written by two structural biologists, Peter Tompa of Vrije Universiteit in Brussels and George Rose of Johns Hopkins University, neither of whom is an intelligent-design advocate. But the paper’s arguments bear so strongly on the design debate, and represent so remarkable a challenge to widely held assumptions about (for instance) the origin of cells, that its effect promises to be far-reaching. As in, revolutionary.

[…]Tompa and Rose draw a number of lessons from their calculations. They argue, first, that any increase in biological realism will only make the Levinthal interactome paradox worse:

Of course, there are additional complicating factors such as alternative splicing, post-translational modifications, non-pairwise macromolecular interactions, incorrect complex formation that is adventitiously stable, and so forth. However, even neglecting such complications, the numbers preclude formation of a functional interactome by trial and error complex formation within any meaningful span of time. This numerical exercise…is tantamount to a proof that the cell does not organize by random collisions of its interacting constituents.But secondly, what they call “the most profound conclusion” from their analysis bears directly on widely held assumptions about the origin of life.

A highly enriched soup of proteins and nucleic acids will never form a functional cell, even if lipid bilayer membranes were provided to help these materials become organized. Indeed, the fully functional contents of a living cell, once the wall or membrane enclosing them has been breached (thus, killing the cell), move irreversibly in the direction of non-living chemistry. Humpty Dumpty, once he cracks, does not reconstitute, but enters what Tompa and Rose call the “zone of chaos,” never to return.

Tompa and Rose have sketched the theoretical basis for why this happens:

[O]ur calculations of combinatorial complexity [show] that the emergent interactome could not have self-organized spontaneously from its isolated protein components. Rather, it attains its functional state by templating the interactome of a mother cell and maintains that state by a continuous expenditure of energy. In the absence of a prior framework of existing interactions, it is far more likely that combined cellular constituents would end up in a non-functional, aggregated state, one incompatible with life…The spontaneous origination of a de novo cell has yet to be observed; all extant cells are generated by the division of pre-existing cells that provide the necessary template for perpetuation of the interactome.

Tompa and Rose spell out other implications of their analysis (e.g., for medicine and synthetic biology), but maybe we’ve piqued your curiosity enough already. This paper deserves your attention. As noted, for a close circle of us at Discovery and Biologic, it’s the most interesting and significant paper we’ve read in years.

Dr. Nelson’s post explains a bit more with pictures.